Entity Details

Primary name Ramp2
Entity type gene
Source Source Link

Details

PrimaryID54409
RefseqGene
SymbolRamp2
Namereceptor (calcitonin) activity modifying protein 2
Chromosome11
Location11 64.47 cM
TaxID10090
Statuslive
SourceGenomegenomic
SourceOriginnatural
CreationDate2000-07-06
ModificationDate2021-06-23

Ontological Relatives

UniProt IDsRAMP2_MOUSE

GO terms

Show/Hide Table
GOName
GO:0001525 angiogenesis
GO:0001570 vasculogenesis
GO:0001605 adrenomedullin receptor activity
GO:0002040 sprouting angiogenesis
GO:0005737 cytoplasm
GO:0006816 calcium ion transport
GO:0006886 intracellular protein transport
GO:0007186 G protein-coupled receptor signaling pathway
GO:0007189 adenylate cyclase-activating G protein-coupled receptor signaling pathway
GO:0007507 heart development
GO:0008217 regulation of blood pressure
GO:0008277 regulation of G protein-coupled receptor signaling pathway
GO:0009986 cell surface
GO:0010628 positive regulation of gene expression
GO:0015026 coreceptor activity
GO:0015031 protein transport
GO:0031623 receptor internalization
GO:0032092 positive regulation of protein binding
GO:0032870 cellular response to hormone stimulus
GO:0034333 adherens junction assembly
GO:0035924 cellular response to vascular endothelial growth factor stimulus
GO:0043116 negative regulation of vascular permeability
GO:0043235 receptor complex
GO:0045766 positive regulation of angiogenesis
GO:0070830 bicellular tight junction assembly
GO:0070831 basement membrane assembly
GO:0072659 protein localization to plasma membrane
GO:0097084 vascular associated smooth muscle cell development
GO:0097643 amylin receptor activity
GO:0097647 amylin receptor signaling pathway
GO:0150057 amylin receptor complex 2
GO:1903143 adrenomedullin receptor complex
GO:1990409 adrenomedullin binding
GO:1990410 adrenomedullin receptor signaling pathway
GO:2000352 negative regulation of endothelial cell apoptotic process
GO:2001214 positive regulation of vasculogenesis

Diseases

Show/Hide Table

Interactions

0 interactions

InteractorPartnerSourcesPublicationsLink