Entity Details

Primary name EFNA1
Entity type gene
Source Source Link

Details

PrimaryID1942
RefseqGene
SymbolEFNA1
Nameephrin A1
Chromosome1
Location1q22
TaxID9606
Statuslive
SourceGenomegenomic
SourceOriginnatural
CreationDate1998-08-10
ModificationDate2021-06-20

Ontological Relatives

UniProt IDsEFNA1_HUMAN

GO terms

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GOName
GO:0000122 negative regulation of transcription by RNA polymerase II
GO:0001525 angiogenesis
GO:0001934 positive regulation of protein phosphorylation
GO:0003180 aortic valve morphogenesis
GO:0003183 mitral valve morphogenesis
GO:0003199 endocardial cushion to mesenchymal transition involved in heart valve formation
GO:0005102 signaling receptor binding
GO:0005576 extracellular region
GO:0005886 plasma membrane
GO:0007267 cell-cell signaling
GO:0007411 axon guidance
GO:0010719 negative regulation of epithelial to mesenchymal transition
GO:0014028 notochord formation
GO:0016477 cell migration
GO:0033628 regulation of cell adhesion mediated by integrin
GO:0034446 substrate adhesion-dependent cell spreading
GO:0043409 negative regulation of MAPK cascade
GO:0043410 positive regulation of MAPK cascade
GO:0043535 regulation of blood vessel endothelial cell migration
GO:0045765 regulation of angiogenesis
GO:0046658 anchored component of plasma membrane
GO:0046875 ephrin receptor binding
GO:0048013 ephrin receptor signaling pathway
GO:0050730 regulation of peptidyl-tyrosine phosphorylation
GO:0050731 positive regulation of peptidyl-tyrosine phosphorylation
GO:0050770 regulation of axonogenesis
GO:0050821 protein stabilization
GO:0061002 negative regulation of dendritic spine morphogenesis
GO:0061098 positive regulation of protein tyrosine kinase activity
GO:0070244 negative regulation of thymocyte apoptotic process
GO:1902004 positive regulation of amyloid-beta formation
GO:1902961 positive regulation of aspartic-type endopeptidase activity involved in amyloid precursor protein catabolic process
GO:1903051 negative regulation of proteolysis involved in cellular protein catabolic process

Diseases

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