Entity Details
| Primary name |
S26A1_HUMAN |
| Entity type |
UniProt |
| Source |
Source Link |
Details
| Accession | Q9H2B4 |
| EntryName | S26A1_HUMAN |
| FullName | Sulfate anion transporter 1 |
| TaxID | 9606 |
| Evidence | evidence at protein level |
| Length | 701 |
| SequenceStatus | complete |
| DateCreated | 2002-03-05 |
| DateModified | 2021-06-02 |
Subcellular Location
Show/Hide Table
| Subcellular Location |
| Basolateral cell membrane |
| Cell membrane |
Domains
Show/Hide Table
| Domain | Name | Category | Type |
| IPR001902 | SLC26A/SulP transporter | Family | Family |
| IPR002645 | STAS domain | Domain | Domain |
| IPR011547 | SLC26A/SulP transporter domain | Domain | Domain |
| IPR018045 | Sulphate anion transporter, conserved site | Site | Conserved site |
| IPR030331 | Solute carrier family 26 member 1 | Family | Family |
| IPR036513 | STAS domain superfamily | Family | Homologous superfamily |
Diseases
Show/Hide Table
| Disease ID | Source | Name | Description |
| 167030 | OMIM | Nephrolithiasis, calcium oxalate (CAON) | A form of nephrolithiasis, a condition in which urinary supersaturation leads to stone formation in the urinary system. Patients manifest acute renal colic with severe pain originating in the flank. Patients with small, non-obstructing stones or those with staghorn calculi may be asymptomatic. The majority of renal calculi contain calcium. CAON is characterized by calcium oxalate kidney stones. The disease is caused by variants affecting the gene represented in this entry. |
Interactions
3 interactions