Entity Details

Primary name ELMO2_HUMAN
Entity type UniProt
Source Source Link

Details

AccessionQ96JJ3
EntryNameELMO2_HUMAN
FullNameEngulfment and cell motility protein 2
TaxID9606
Evidenceevidence at protein level
Length720
SequenceStatuscomplete
DateCreated2003-05-16
DateModified2021-06-02

Ontological Relatives

GenesELMO2

GO terms

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GOName
GO:0005829 cytosol
GO:0006915 apoptotic process
GO:0007015 actin filament organization
GO:0016020 membrane
GO:0017124 SH3 domain binding
GO:0030971 receptor tyrosine kinase binding
GO:0038096 Fc-gamma receptor signaling pathway involved in phagocytosis
GO:0048010 vascular endothelial growth factor receptor signaling pathway
GO:0048870 cell motility
GO:0060326 cell chemotaxis
GO:0098609 cell-cell adhesion

Subcellular Location

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Subcellular Location
Cytoplasm
Membrane

Domains

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DomainNameCategoryType
IPR001849 Pleckstrin homology domainDomainDomain
IPR006816 ELMO domainDomainDomain
IPR011989 Armadillo-like helicalFamilyHomologous superfamily
IPR011993 PH-like domain superfamilyFamilyHomologous superfamily
IPR016024 Armadillo-type foldFamilyHomologous superfamily
IPR024574 Domain of unknown function DUF3361DomainDomain
IPR030713 Engulfment and cell motility protein 2FamilyFamily

Diseases

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Disease IDSourceNameDescription
606893 OMIMVascular malformation, primary intraosseous (VMOS)A rare malformation characterized by non-neoplastic severe expansions of blood vessels, usually seen in the vertebral column and in the skull. The most commonly affected bones in the skull are the mandible and the maxilla, and life-threatening bleeding after a simple tooth extraction is frequently observed. Inheritance is autosomal recessive. The disease is caused by variants affecting the gene represented in this entry.

Interactions

3 interactions