Entity Details

Primary name NU155_HUMAN
Entity type UniProt
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Details

AccessionO75694
EntryNameNU155_HUMAN
FullNameNuclear pore complex protein Nup155
TaxID9606
Evidenceevidence at protein level
Length1391
SequenceStatuscomplete
DateCreated1999-07-15
DateModified2021-06-02

Ontological Relatives

GenesNUP155

GO terms

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GOName
GO:0000972 transcription-dependent tethering of RNA polymerase II gene DNA at nuclear periphery
GO:0005635 nuclear envelope
GO:0005829 cytosol
GO:0006110 regulation of glycolytic process
GO:0006405 RNA export from nucleus
GO:0006406 mRNA export from nucleus
GO:0006409 tRNA export from nucleus
GO:0006606 protein import into nucleus
GO:0006998 nuclear envelope organization
GO:0007084 mitotic nuclear membrane reassembly
GO:0016020 membrane
GO:0016032 viral process
GO:0016925 protein sumoylation
GO:0017056 structural constituent of nuclear pore
GO:0019058 viral life cycle
GO:0019083 viral transcription
GO:0031965 nuclear membrane
GO:0036228 protein localization to nuclear inner membrane
GO:0043657 host cell
GO:0044611 nuclear pore inner ring
GO:0060964 regulation of gene silencing by miRNA
GO:0075733 intracellular transport of virus
GO:0086014 atrial cardiac muscle cell action potential
GO:1900034 regulation of cellular response to heat

Subcellular Location

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Subcellular Location
Nucleus
Nucleus membrane

Domains

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DomainNameCategoryType
IPR004870 Nucleoporin, Nup155-likeFamilyFamily
IPR007187 Nucleoporin, Nup133/Nup155-like, C-terminalDomainDomain
IPR014908 Nucleoporin, Nup133/Nup155-like, N-terminalDomainDomain
IPR042533 Nucleoporin, Nup155-like, C-terminal, subdomain 1FamilyHomologous superfamily
IPR042537 Nucleoporin, Nup155-like, C-terminal, subdomain 2FamilyHomologous superfamily
IPR042538 Nucleoporin, Nup155-like, C-terminal, subdomain 3FamilyHomologous superfamily

Diseases

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Disease IDSourceNameDescription
615770 OMIMAtrial fibrillation, familial, 15 (ATFB15)A familial form of atrial fibrillation, a common sustained cardiac rhythm disturbance. Atrial fibrillation is characterized by disorganized atrial electrical activity and ineffective atrial contraction promoting blood stasis in the atria and reduces ventricular filling. It can result in palpitations, syncope, thromboembolic stroke, and congestive heart failure. The disease is caused by variants affecting the gene represented in this entry.