Entity Details
Primary name |
ALX3_HUMAN |
Entity type |
UniProt |
Source |
Source Link |
Details
Accession | O95076 |
EntryName | ALX3_HUMAN |
FullName | Homeobox protein aristaless-like 3 |
TaxID | 9606 |
Evidence | evidence at transcript level |
Length | 343 |
SequenceStatus | complete |
DateCreated | 2001-04-27 |
DateModified | 2021-06-02 |
GO terms
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GO | Name |
GO:0000785 | chromatin |
GO:0000977 | RNA polymerase II transcription regulatory region sequence-specific DNA binding |
GO:0000981 | DNA-binding transcription factor activity, RNA polymerase II-specific |
GO:0005634 | nucleus |
GO:0006357 | regulation of transcription by RNA polymerase II |
GO:0007389 | pattern specification process |
GO:0035115 | embryonic forelimb morphogenesis |
GO:0035116 | embryonic hindlimb morphogenesis |
GO:0042981 | regulation of apoptotic process |
GO:0048701 | embryonic cranial skeleton morphogenesis |
GO:1990837 | sequence-specific double-stranded DNA binding |
Subcellular Location
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Subcellular Location |
Nucleus |
Domains
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Domain | Name | Category | Type |
IPR001356 | Homeobox domain | Domain | Domain |
IPR009057 | Homeobox-like domain superfamily | Family | Homologous superfamily |
IPR017970 | Homeobox, conserved site | Site | Conserved site |
IPR033211 | Homeobox protein aristaless-like 3 | Family | Family |
Diseases
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Disease ID | Source | Name | Description |
136760 | OMIM | Frontonasal dysplasia 1 (FND1) | The term frontonasal dysplasia describes an array of abnormalities affecting the eyes, forehead and nose and linked to midfacial dysraphia. The clinical picture is highly variable. Major findings include true ocular hypertelorism; broadening of the nasal root; median facial cleft affecting the nose and/or upper lip and palate; unilateral or bilateral clefting of the alae nasi; lack of formation of the nasal tip; anterior cranium bifidum occultum; a V-shaped or widow's peak frontal hairline. The disease is caused by variants affecting the gene represented in this entry. |
Interactions
0 interactions
Interactor | Partner | Sources | Publications | Link |