Publication Details

Pubmed12006497
Title: Signaling disrupts mSin3A binding to the Mad1-like Sin3-interacting domain of TIEG2, an Sp1-like repressor.
Year: 2002
DOI: https://doi.org/10.1093/emboj/21.10.2451
First Author: Ellenrieder
Experimental setup: affinity chromatography technology (Biogrid)
in vitro (Hprd)
in vivo (Hprd)
Throughput: Low Throughput (Biogrid)

Interactions

2 interactions
InteractorPartnerSourcesLink
KLF11_HUMANSIN3A_HUMANBioGRID, HPRDdetails
MK01_HUMANKLF11_HUMANHPRDdetails